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1 MAIT cells are imprinted by the microbiota in early life and promote tissue repair
2 Rejection of benign melanocytic nevi by nevus-resident CD4+ T cells
3 Absence of mucosal-associated invariant T cells in a person with a homozygous point mutation in MR1
4 A divergent transcriptional landscape underpins the development and functional branching of MAIT cells
5 CD4+ and CD8+ TCRβ repertoires possess different potentials to generate extraordinarily high-avidity T cells
6 Absence of MHC class II on cDC1 dendritic cells triggers fatal autoimmunity to a cross-presented self-antigen